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Prime AGE Defense Formula

Price  $47.00
Cashback$0.94
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Prime AGE Defense Formula was created to help you feel healthy and vibrant as you age.When excess sugar is consumed, it can bond with protein in a process called glycation, resulting in the formation of advanced glycation end products (AGEs), which can...
Single Bottle (30 Servings)
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Prime AGE Defense Formula

Prime AGE Defense Formula was created to help you feel healthy and vibrant as you age.

When excess sugar is consumed, it can bond with protein in a process called glycation, resulting in the formation of advanced glycation end products (AGEs), which can cause problems such as premature wrinkles and organs that don't operate as efficiently as they could.

Most competitors' products only help to reduce the formation of advanced glycation end products, but do not work to support your body's normal ability to remove AGEs through healthy circulation. Prime AGE Defense Formula offers complete support by reducing the formation of AGEs and supporting the body's ability to remove AGEs.

The blend of ingredients in Prime AGE Defense Formula supports numerous areas of health associated with aging, including skin health, circulation, cell function, arterial health, DNA and RNA.

Prime AGE Defense Formula is one of the must-have healthy aging products in the Prime ant-aging supplement lineup to help you feel healthy and vibrant as you age.


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FAQ
  • Beltramo, E., et al. Effects of thiamine and benfotiamine on intracellular glucose metabolism and relevance in the prevention of diabetic complications. Acta Diabetologica. 45(3): 131-141, 2008.
  • Berrone, E., et al. Regulation of intracellular glucose and polyol pathway by thiamine and benfotiamine in vascular cells cultured in high glucose. The Journal of Biological Chemistry. 281(14): 9307-9313, 2006.
  • Cameron, N., et al. Inhibitors of advanced glycation end product formation and neurovascular dysfunction in experimental diabetes. Annals of the New York Academy of Sciences. 1043: 784-792, 2005.
  • Dugoua, JJ., et al. From type 2 diabetes to antioxidant activity: a systematic review of the safety and efficacy of common and cassia cinnamon bark. Canadian Journal of Physiology and Pharmacology. 85(9): 837-847, 2007.
  • Hammes, HP., et al. Benfotiamine blocks three major pathways of hyperglycemic damage and prevents experimental diabetic retinopathy. Nature Medicine. 9(3): 294-299, 2003.
  • Head, K. Peripheral neuropathy: pathogenic mechanisms and alternative therapies. Alternative Medicine Review. 11(4): 294-329, 2006.
  • Lee, L., et al. Possible differential induction of phase 2 enzyme and antioxidant pathways by American ginseng, Panax quinquefolius. Journal of Clinical Pharmacology. 48(5): 599-609, 2008.
  • Marchetti, V., et al. Benfotiamine counteracts glucose toxicity effects on endothelial progenitor cell differentiation via Akt/FoxO signaling. Diabetes. 55(8): 2231-2237, 2006.
  • Nakayama, M., et al. Suppression of n-(carboxymethyl)lysine generation by the antioxidant n-acetyl cysteine. Peritoneal Dialysis International. 19: 207-210, 1999.
  • Nandhini, A., et al. Stimulation of glucose utilization and inhibition of protein glycation and AGE products by taurine. Acta Physiologica Scandinavica. 181(3): 297-303, 2004.
  • Nandhini, A., et al. Taurine prevents collagen abnormalities in high fructose-fed rats. Indian Journal of Medical Research. 122(2): 171-177, 2005.
  • Nandhini, A., et al. Taurine prevents fructose-diet induced collagen abnormalities in rat skin. Journal of Diabetes and Its Complications. 19(5): 305-311, 2005.
  • Peng, X., et al. Cinnamon bark proanthocyanidins as reactive carbonyl scavengers to prevent the formation of advanced glycation end products. Journal of Agricultural and Food Chemistry. 56(6): 1907-1911, 2008.
  • Saraswat, M., et al. Prevention of non-enzymic glycation of proteins by dietary agents: prospects for alleviating diabetic complications. British Journal of Nutrition. 101(11): 1714-1721, 2009.
  • Schmid, U., et al. Benfotiamine exhibits direct antioxidant capacity and prevents induction of DNA damage in vitro. Diabetes/Metabolism Research and Reviews. 24(5): 371-377, 2008.
  • Shan, B., et al. Antioxidant capacity of 26 spice extracts and characterization of their phenolic constitutes. Journal of Agricultural and Food Chemistry. 53(20): 7749-7759, 2005.
  • Sheng, X., et al. Improved insulin resistance and lipid metabolism by cinnamon extract through activation of peroxisomes proliferator-activated receptors. PPAR Research. Article ID 581348, 2008.
  • Tarallo, S., et al. Effects of high glucose and thiamine on the balance between matrix metalloproteinases and their tissue inhibitors in vascular cells. Acta Diabetologica. 2009.
  • Thomas, M., et al. The role of AGEs and AGE inhibitors in diabetic cardiovascular disease. Current Drug Targets. 6(4): 453-474, 2005.
  • Vuksan, V., et al. Konjac-mannan and American ginseng: emerging alternative therapies for type 2 diabetes mellitus. Journal of the American College of Nutrition. 20(5): 370S-380S, 2001.
  • Wang, CZ., et al. Commonly used antioxidant botanicals: active constituents and their potential role in cardiovascular disease. American Journal of Chinese Medicine. 35(4): 543-558, 2007.
  • Wu, Z., et al. American ginseng modulates pancreatic beta cell activities. Chinese Medicine. 2(11): 1-5, 2007.
  • Yin, J., et al. Traditional Chinese medicine in treatment of metabolic syndrome. Endocrine, Metabolic & Immune Disorders Drug Targets. 8(2): 99-111, 2008.
 
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